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These results suggest that overall the p16 ink4a gene is more responsible in inducing cellular senescence and tumor suppression in human cells interestingly the ink4aarf locus is known to be repressed by the polycomb proteins b lymphoma momlv insertion region 1 bmi1 and related family members in both human and mouse cellscellular senescence and tumor suppression it is now clear that cellular senescence is a crucial anticancer mechanism that prevents the growth of cells at risk for neoplastic transformation the stimuli that elicit a senescence response all have the potential to initiate or promote carcinogenesiscellular senescence and tumor suppression collects a number of chapters from leaders in the field to review the molecular basis of senescence and its physiological functions with a particular emphasis on the role of senescence in tumor suppressioncellular senescence and tumor suppression collects a number of chapters from leaders in the field to review the molecular basis of senescence and its physiological functions with a particular emphasis on the role of senescence in tumor suppression en schema it was reported that oncogenic ras induces dna damage signaling and the activation of the cell cycle checkpoint which are critical for the cellular senescence and tumor suppression phenotypes 4042 therefore we have examined h2ax foci formation a dna damage response marker in tumors in all the genotypes h2ax foci were detectable

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